Liver Innovation Platform For Biotech Teams, Startup and Investors - The Liver Investor Network

The Liver Investor Network is an investor and partnering platform devoted to providing private and public biotechnology and life sciences companies with an opportunity to present to, and meet with, investors and pharmaceutical executives in one place during Jerusalem meeting.

Come and present, discuss, exchange and initiate a collaboration with Israeli and international leaders and actors of liver world.

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The Microbiome and Liver Disease – Towards a new understanding

The liver receives the majority of its blood supply from the portal vein, and thus represents the first pass organ for food antigents and products derived from the intestinal microbiota. The team of Dr Christoph Thaiss, from Weizmann Institute of Science, Israel has recently demonstrated that this microbiota-liver axis plays a fundamental role in the initiation and progression of hepatic disease.

Vitamin E and Diabetic Nephropathy; Lessons and Clinical Implications

In diabetes mellitus there is an increase in oxygen radical formation due to glucose auto oxidation, the formation of advanced glycosylation end products, and metabolic stress. Epidemiologic studies suggest that vitaminE supplementation might decrease the risk of developing cardiovascular disease, others showed increased risk of cardiac death with the vitamin E treatment.

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Stellate Cells in Hepatic Fibrogenesis and Protection by a Novel Modulator

Dr Raj Lakshman, The George Washington University, Washington, D.C., USA will talk about Stellate Cells in Hepatic Fibrogenesis and Protection by a Novel Modulator during Targeting Liver Diseases World Congress.

According Dr Lakshman: "Hepatic Stellate Cells (HSC) are the main fibrogenic cells of the liver.

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Liver repopulation following toxic liver injury - which genes have most impact?

Prior research on hepatocyte proliferation has focused on the easily accessible paradigm of partial hepatectomy, which does not model the regenerative response of hepatocytes following toxic injury, such as occurs following alcohol or acetaminophen poisoning.

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